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  • In exchange for a two or three-year (for Intramural General Research) commitment to your research career, NIH will repay up to $35,000 per year of your qualified educational debt. See NIH Loan Repayment Programs

Research in NIAMS Labs

Updated November 5, 2014

Photo of Dr. John O'Shea, NIAMS Scientific Director

Welcome to NIAMS Labs from Dr. John O'Shea, NIAMS Scientific Director

As you will see, our program comprises scientists, physicians, nurses, trainees and administrators who work together to solve basic scientific problems and devise new treatments for arthritis, musculoskeletal and skin diseases.

Image showing damage (in color) to tooth enamel

Research on Keratins Reveals Unexpected Link Between Hair Disorders and Dental Decay

Keratins are proteins that are key structural components of hair, nails, and the skin’s outer layer. Now, new research has shown that certain types of hair keratin also help form dental enamel, the tough outer covering of teeth.

Photo of Dr. John O'Shea

NIAMS' Scientific Director Elected to Institute of Medicine

John J. O’Shea, M.D., has been elected to membership in the Institute of Medicine (IOM) of the National Academy of Sciences. He has "advanced the field of immunology by translating his discoveries into approaches that improve human health," says NIAMS Director Stephen I. Katz, M.D., Ph.D.

Photos of Drs. Laurence, Kanno and O'Shea.

Three NIAMS Researchers Named "Most Influential"

Drs. John O’Shea, Yuka Kanno and Arian Laurence have been included by Thomson Reuters in The World’s Most Influential Scientific Minds 2014 report, in immunology, joining the ranks of researchers who are "publishing work that their peers recognize as vital to the advancement of their science" and "on the cutting edge of their fields."

Schematic of the increased neutrophil extracellular trap (NET) formation in SLE

The Kaplan Lab Shows an Additional Proatherogenic Mechanism in SLE

This work identifies neutrophils as important inducers of lipoprotein oxidation, suggesting an additional mechanism for premature vascular disease in autoimmune disorders.