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Research in NIAMS Labs
Updated April 7, 2016
NIAMS Scholar Receives K99/R00 Career Transition Award
Congratulations to Dr. Vivek Thumbigere Math, a clinical fellow in the Laboratory of Oral Connective Tissue Biology, who received a NIH Pathway to Independence Award (K99/R00), funded by NIAMS. This award will support Dr. Thumbigere Math and colleagues’ research to better understand the genetic defects and immunopathological mechanisms contributing to inflammatory-mediated tissue destruction in periodontal disease.
NIAMS Researchers Present at Keystone Symposia Conference
Dr. Eric Hanson, head of the Immunodeficiency and Inflammation Unit, and Ms. Claire Tocheny, postbaccaluareate fellow, recently gave talks at the NF-κB and MAP Kinase Signaling in Inflammation Conference. This meeting was part of the Keystone Symposia 2015-2016 Conference Series and took place March 13–17, 2016, in Whistler, British Columbia.
NIAMS Scholar Receives K22 Career Transition Award
Congratulations to Dr. Scott Canna, head of the Autoinflmmatory Pathogenesis Unit, who received a K22 Career Transition Award, funded by NIAID. This award will support Dr. Canna and colleagues’ research to better understand the mechanism driving their recent discovery that NLRC4 inflammasome mutations cause a life-threatening inflammatory state called macrophage activation syndrome.
NIAMS Alum Receives Prestigious Sloan Research Fellowship
Congratulations to Dr. Golnaz Vahedi who was selected as a 2016 Alfred P. Sloan Research Fellow in Computational & Evolutionary Molecular Biology. Dr. Vahedi completed her postdoctoral training in the Molecular Immunology and Inflammation Branch. She is currently an Assistant Professor with the Department of Genetics, University of Pennsylvania School of Medicine.
Anti-interferon Autoantibodies in Systemic Lupus Erythematosus
A recently published paper from Dr. Sarthak Gupta, NIAMS Scholar in the Rheumatology Training Program, evaluates the role of autoantibodies in lupus and other systemic autoimmune diseases. Researchers found that lupus patients have an excess of autoantibodies against signaling proteins called interferons. These anti-interferon autoantibodies are a potential therapeutic target for lupus.
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- Superior Athleticism: Is It in Your Genes?
- NIH Researchers Reveal Link Between Powerful Gene Regulatory Elements and Autoimmune Diseases